Spinal stenosis is the narrowing of the spinal canal in the lumbar (lower back) region, resulting in pressure on the lumbar nerves (cauda equina). Spinal stenosis usually becomes symptomatic after the age of 50. It is caused by degeneration of the spine (with rare exceptions in patients born with a congenitally narrow canal). In most patients, symptom onset begins with an acute event, such as a lumbar injury or a herniated disc.
Symptoms of spinal stenosis include:

Diagnosis is based on a combination of medical history, which usually includes difficulty walking distances (walking interrupted by the need to sit, i.e., intermittent claudication), and physical examination findings.
During the physical exam, patients may show weakness in certain nerves, affecting specific muscle groups in the legs. They may also show limited motion or reduced sensation. A thorough exam should always include the hips (which often mimic spinal stenosis) and the lower limb blood vessels, since vascular stenosis of the legs can produce similar symptoms.
Spinal stenosis is a progressive disease for two reasons:
Walking distance gradually decreases, and the longer nerve compression persists, the less recovery of walking ability is possible.
Conservative treatment is not curative, only symptomatic relief.
The goal of surgery is to widen the spinal canal and decompress the nerves.
Spinal fusion is indicated only in specific cases:
During fusion surgery, after decompression, screws and rods are used to stabilize the vertebrae. Bone grafts are then applied to achieve permanent fusion.
At home:
Activity progression depends on the extent of surgery, previous activity level, and type of surgery:
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